Scientists from A*STAR Institute of Molecular and Cell Biology (A*STAR IMCB) have identified why certain lung cancer cells ...

A hidden clue may explain why some mutated cells become cancerous and others don’t: how fast they divide. A new study from researchers at Sinai Health in Toronto reveals that the total time it takes ...

A hidden communication network between brain cells and glioblastoma tumors may be key to slowing this aggressive cancer.

The cancer gene MYC camouflages tumours by suppressing alarm signals that normally activate the immune system. This finding ...

Johns Hopkins Medicine scientists say they have found a pattern of so-called epigenetic "marks" in a transition state between normal and pancreatic cancer cells in mice, and that the normal cells may ...

Some cancer cells don't die; they go quiet, like seeds lying dormant in the soil. These "sleeper cells," scattered throughout the body, can stay inactive for years. But when the body faces a ...

Scientists have recently been learning more about the importance of small bits of circular genetic material known as extrachromosomal DNA (ecDNA). These little circles of DNA can hitch a ride with ...

Johns Hopkins Medicine scientists say they have found a pattern of so-called epigenetic "marks" in a transition state between normal and pancreatic cancer cells in mice, and that the normal cells may ...

The research, published today in Nature Communications, focuses on how cancer cells keep their chromosomes intact so they can continue dividing relentlessly. Normally, every time a cell divides, the ...

Scientists from A*STAR Institute of Molecular and Cell Biology (A*STAR IMCB) have identified why certain lung cancer cells become highly resistant to treatment after developing mutations in a key gene ...